MODULATION OF EPILEPTIC ACTIVITY IN RATS: FOCUS ON SLEEP, PHYSICAL EXERCISE AND NITRIC OXIDE-MEDIATED NEUROTRANSMISSION IN A MODEL OF HOMOCYSTEINE THIOLACTONE SEIZURES

  • Dragan Hrncic 1Laboratory of Neurophysiology, Institute of Medical Physiology “Richard Burian”, Faculty of Medicine, University of Belgrade, 11000 Belgrade, Serbia

Abstract


Epilepsy is a chronic neurological disorder characterized by recurrent epileptic seizures. Understanding of its mechanisms of initiation and development, as well as modulating factors, are of great scientific interest. Experimental models of epilepsy are useful in understanding these mechanisms.  

Homocysteine, an amino acid endogenously generated in the body, together with its reactive metabolite homocysteine thiolactone (HCT), is recognized as a risk factor for variety of diseases. HCT-induced seizures are model of generalized epilepsy in which coexistence of two types of epileptic activity were documented. Complex interplay between sleep and epilepsy is still only poorly understood. Also, relationship between physical exercise and epilepsy is quite intriguing, especially underlying mechanism involved in this relationship. The role of nitric oxide (NO)-mediated neurotransmission in development of the epileptic activity is highly contradictor in the existing scientific literature.

In this review article we addressed modulation of epileptic activity in rats focusing sleep, physical activity and NO-mediated signaling. Firstly, we considered concepts of experimental models of epileptic activity with unique features of HCT seizures. Secondly, modulating effects of sleep and regular physical exercise training  on epileptic activity, along with works from the authors, are discussed. Finally, the anticonvulsive effects of NO derived by nNOS and iNOS in HCT seizures are reviewed.

Key words: homocysteine, seizures, sleep, physical activity, nitric oxide

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Published
2014/04/14
Section
Review Paper